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Università degli Studi di Roma 'Tor Vergata'


Neuroscienze

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Prof. Pietro Calissano


Pubblicazioni back

  1. Increased amyloidogenic secretion in cerebellar granule cells undergoing apoptosis. P.N.A.S. USA, 95 :1247-1252, 1998;
  2. Neuronal apoptosis in rats is accompanied by rapid impairment of cellular respiration and is preveted by scavengers of reactive oxygen species. Neurosci. Lett. 245 : 127-130, 1998;
  3. Tau cleavage and dephosphhorylation in cerebellar granule neurons undergoing apoptosis. J. Neurosci. 18 :7061-7074, 1998;
  4. Adenosine and ADP prevent apoptosis in cultured rat cerebellar granule cells. Brain Res. 27807, 1998;
  5. Early release and subsequent caspase-mediated degradation of cytochrome c in apoptotic cerebellar granule cells. FEBS Lett. 1999 Aug 20;457(1):126-30;
  6. Glutamate neurotoxicity in rat cerebellar granule cells involves cytochrome c release from mitochondria and mitochondrial shuttle impairment. J Neurochem. 1999 Jul;73(1):237-46;
  7. Cytochrome c is released from mitochondria in a reactive oxygen species (ROS)-dependent fashion and can operate as a ROS scavenger and as a respiratory substrate in cerebellar neurons undergoing excitotoxic death. J Biol Chem. 2000 Nov 24;275(47):37159-66;
  8. Endogenous APP derivatives oppositely modulate apoptosis through an autocrine loop. Neuroreport. 2000 May 15;11(7):1375-9;
  9. Proteasome involvement and accumulation of ubiquitinated proteins in cerebellar granule neurons undergoing apoptosis. J Neurosci. 2000 Jan 15;20(2):589-99;
  10. Granule neuron DNA damage following deafferentation in adult rats cerebellar cortex: a lesion model. Neuroscience. 2000;95(1):163-71.

 

Coordinatore:
Prof. Giorgio Bernardi


06/72596004 back bernardi@uniroma2.it