|
- Increased amyloidogenic secretion in cerebellar granule cells
undergoing apoptosis. P.N.A.S. USA, 95 :1247-1252, 1998;
- Neuronal apoptosis in rats is accompanied by rapid impairment of
cellular respiration and is preveted by scavengers of reactive oxygen species.
Neurosci. Lett. 245 : 127-130, 1998;
- Tau cleavage and dephosphhorylation in cerebellar granule neurons
undergoing apoptosis. J. Neurosci. 18 :7061-7074, 1998;
- Adenosine and ADP prevent apoptosis in cultured rat cerebellar granule
cells. Brain Res. 27807, 1998;
- Early release and subsequent caspase-mediated degradation of
cytochrome c in apoptotic cerebellar granule cells. FEBS Lett. 1999 Aug
20;457(1):126-30;
- Glutamate neurotoxicity in rat cerebellar granule cells involves
cytochrome c release from mitochondria and mitochondrial shuttle impairment. J
Neurochem. 1999 Jul;73(1):237-46;
- Cytochrome c is released from mitochondria in a reactive oxygen
species (ROS)-dependent fashion and can operate as a ROS scavenger and as a respiratory
substrate in cerebellar neurons undergoing excitotoxic death. J Biol Chem. 2000 Nov
24;275(47):37159-66;
- Endogenous APP derivatives oppositely modulate apoptosis through an
autocrine loop. Neuroreport. 2000 May 15;11(7):1375-9;
- Proteasome involvement and accumulation of ubiquitinated proteins in
cerebellar granule neurons undergoing apoptosis. J Neurosci. 2000 Jan 15;20(2):589-99;
- Granule neuron DNA damage following deafferentation in adult rats
cerebellar cortex: a lesion model. Neuroscience. 2000;95(1):163-71.
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