Neuroscienze

Prof. Marco Trabucchi

1. Isoniazid-induced inhibition of GABAergic Transmission Enhances Neurosteroid Content in the rat Brain. Neuropharmacology 1996 ; 35:35:1299-1305;
2. Exsecutive Dysfunction in Early Alzheimer's Disease. J. Neurol. Neurosurg. Psychiatry 1996;60:91-93;
3. Fibroblasts of Patients Affected by Down's Syndrome Oversecrete Amyloid Precursor Protein and are hiporesponsive to Protein Kinase C. Stimulation. Neurology 1996;47: 1069-1075;
4. The effects of inhibitors of GABAergic Transmission and Stress on Brain and Plasma Allopregnanolone Concentrations. Br. J. Pharmacolofy 1997;120:1582-1588;
5. Clinical Rationale of Genetic testing in Dementia. J. Neurol. Neurosurg. Psychiatry. 1997;62:217-221;
6. Surface Dyslexia in an Italian Patient with Semantic Dementia. Neurocase 1997; 3:307-312;
7. Differential level of Platelet Amyloid Precursor Protein Isoforms. An Early Marker for Alzheimer Disease. Arch. Neurol. 1998; 55:1195-1200;
8. Peripheral Markers in Testing Pathophysiological Hypothesis and Diagnosing Alzheimer's Disease. FASEB J. 1998;12:17-34;
9. Bradykinin-Induced Amyloid Precursor Protein Secretion: a Protein Kinase C- Independent Mechanism that is not Altered in Fibroblasts from Patients with Sporadic Alzheimer's Disease. Biochem. J. 1998;330:1271-75;
10. Validity of Direct Assessment of Functional Status as a Tool for measuring Alzheimer's Disease severity. Age Ageing 1998; 27:615-622.