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Conference:
“AIDS and infectious diseases in the age of change”.
December 1 - 1998 , IRCCS “L. Spallanzani”, Rome.
The conference was held with the participation of the Minister
of Health R. Bindi, the Director of the National Institute of Health Prof. G. Benagiano, the President of the World Foundation for AIDS Research & Prevention Prof.
L. Montagnier, the High Commissioner of IRCCS “L. Spallanzani” Dr. E. Melaragno and the President of C.N.R. “Tor Vergata Area“ Prof. E. Garaci.
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Seminary:
“AIDS prevention and scientific communication”
University of Rome “Tor Vergata”, july 21, 1998
Prof.V. Colizzi, Ph.D., M.D., Professor of Immunology and Molecular Pathology, University of Rome “Tor Vergata” - Director of ICAERI
Seminary:
“HIV spread in the world”
University of Rome “Tor Vergata”, july 21, 1998
Dr. G.Ippolito, Institute for Infectious Diseases “L.Spallanzani”- Scientific Director
Seminary:
“The immune system in AIDS”
University of Rome “Tor Vergata”, july 21, 1998
Dr. Fabrizio Poccia, Ph.D., ICAERI Staff
Seminary:
“Allergenic plants”
IRCCS “L. Spallanzani”, january 21, 1999
Prof. M. Grilli Caliola, Department of Biology, University of Rome “Tor Vergata”
Seminary:
“Mechanisms of immune-recognition by natural killer”
IRCCS “L. Spallanzani”, january, 1999
Prof. M. López-Botet, Hospital De la Princesa Servicio de Immunologia, Madrid, Spain.
Seminary:
“Glycolipidic self: a new class of autoantigens”
IRCCS “L. Spallanzani”, may, 1999
Dr. G.De Libero, Experimental Immunology Department of Research, Kantonsspital Basel, Switzerland
Seminary:
“Purinergic receptors and apoptosis in mycobacterial diseases”
IRCCS “L. Spallanzani”, june, 1999
Dr. Giorgio Mancino, University of Rome, “Tor Vergata”
Seminary:
“Transgenic mice as a model for the study of infectious diseases”
IRCCS “L. Spallanzani”, october, 1999
Dr. Lucia Mori, Experimental Immunology, Department of Research, Kantonsspital Basel, Switzerland
Thesis:
University of Rome “Tor Vergata” , 15 july 1999
Flavio Di Pietro, Thesis in Immunology: “Modulation of the expression of HLA class Ib molecules during HIV-1 infection and immune-recognition by natural killer cells”.
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